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TB-500 is a synthetic fraction of the protein thymosin beta-4, which is present in virtually all human and animal cells. It consists of 43 amino acids and the gene, TMSB4X encodes it.
Thymosin beta 4 is produced in high concentrations in damaged tissues. TB-500 purports to promote endothelial cell differentiation, angiogenesis, keratinocyte migration, collagen deposition, and decreased inflammation. The healing effect of TB-500 is observed in tendons, ligaments, muscle, skin, heart, and eyes.
After an injury, thymosin beta 4 is released by platelets, macrophages, and many other cell types, to protect cells and tissues from further damage and reduce apoptosis, inflammation and microbial growth. Thymosin beta 4 binds to actin and promotes cell migration, which includes the mobilization, migration, and differentiation of stem/progenitor cells, which form new blood vessels and regenerate the tissue. Thymosin beta 4 also decreases the number of myofibroblasts in wounds, resulting in decreased scar formation and fibrosis.
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The pentapeptide Ipamorelin is classified as a GHRP (growth hormone-releasing peptide), which is similar to GHRP-2, GHRP-6, and Hexarelin in stimulating growth hormone (GH) in animal test subjects.
It is the mildest alternative to GHRP-2 or GHRP-6, because it has similar effects on test subjects, and doesn’t come with as many of the side effects which are noted when during trials of other Growth Hormone Releasing Peptides.
Surprisingly, Ipamorelin has not been shown to release adrenocorticotropic hormone (ACTH), prolactin or cortisol in levels significantly different from those observed, following GHRH stimulation.
Іраmоrеlіn mіmісѕ thе GН rеlеаѕіng рерtіdе (GНRР) ghrеlіn bу bіndіng tо thе GНRР rесерtоrѕ аnd асtіvаtіng GН rеlеаѕе.
Іn соntrаѕt tо оthеr соmроundѕ thаt асtіvаtе thе ріtuіtаrу GН ѕуѕtеm, ѕuсh аѕ GHRP-2 and -6, Iраmоrеlіn іѕ mоrе ѕеlесtіvе іn іtѕ асtіvаtіоn. It іѕ аlѕо mеtаbоlіzеd ѕlоwеr thаn оthеr GНRРѕ, mаkіng іt mоrе ѕtаblе аnd thеrеbу еffесtіvе fоr lоngеr.
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CJC-1295 is a synthetic analog of the Growth Hormone-Releasing Hormone. With a substitution of four amino acids, it is more resistant to proteolytic inactivation by dipeptidyl peptidase IV (DPP-IV). When combined with Drug Affinity Complex (DAC), it prolongs its half-life, as well as the duration of action in test subjects.
Studies in animals (rats, pigs, and dogs) show that serum IGF-I levels increase several days after a single administration of CJC-1295 with DAC. It sustained a dose-dependent stimulation of serum GH and IGF-I levels when injected into healthy subjects at intervals of 1-2 weeks.
It increases plasma levels of growth hormone from somatotropic cells in the pituitary gland of animal test subjects, without altering its pulsation. It doesn’t inhibit the effects of somatostatin.
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Growth Hormone peptide Fragment 176-191 is also known as HGH Frag 176-191 and AOD9604. It is the modified form of the tail end of the Human Growth Hormone (HGH) molecule, known as the C terminal region. It constitutes less than ten percent of the total size of the HGH molecule, which is the fat-reducing part. It doesn’t affect growth or insulin resistance.
It mimics the way natural Growth Hormone regulates fat metabolism but is 12.5 times stronger for weight loss. It does this without affecting blood sugar or growth observed with unmodified Growth Hormone in animal test subjects. Fragment 176-191 has been shown in studies to release fat specifically from obese fat cells but not from lean ones, reduces new fat accumulation in all fat cells, and enhances fat burning.
It has a short half-life with peak concentrations occurring around 15 minutes and no longer than 60 minutes after administration.
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